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What is Alzheimer's Disease?

Alzheimer's disease impairs the brain's intellectual functions (memory, orientation, calculation), but at first, largely spares those parts of the brain that control sensation and movement. The memory often is affected early, and gradually deteriorates, causing impaired judgment and other problems that may affect a person's ability to perform normal daily activities. Although Alzheimer's usually begins later in life, generally after age 60, rarely it affects people as young as age 30.
For information on causes, diagnosis, and resources click here.

See also: Aging

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Research News

Featuring Harvard Medical School and Affiliated Hospitals

Stroke and Head Injury can increase risk of Alzheimer's.

Imaging Compound identifies amyloid-beta in human brain, may track treatment results.

Study Finds Common Anesthetic may induce cell death and generation of disease-related protein

The Relationship Between Delirium
and Dementia

Novel Pathway Regulates Timing of Brain-cell Development
Findings may aid n understanding Alzheimer's.

Potential Vaccine Improves Learning and Memory Deficits in Mice
Might give the brain it's best chance at recovery.

Damaged Genes in Aging Human Brain Provide Clues to Cognitive Decline

Possible Link Between Diabetes and Alzheimer's Disease
New study suggests insulin resistance in brain cells can affect brain function.

New Targets for Treatments
Toxic protein build up can lead to disease.

Enzyme Elevated in Key Brain Areas Elevated enzymes build toxic brain plaques.

>More Research News

Research Stories

From the Harvard University Gazette

Finding the Start of Alzheimer’s Disease

Possible Mechanism for Link Between Diabetes and Alzheimer's
Insulin resistance observed in brain cells.

Alzheimer's Vaccine Looks Promising
A nose drop vaccine has successfully treated Alzheimers in mice.

>More from HU Gazette on Alzheimer's

Scientific Reports

From HMS Faculty newsletter Focus

Traditional Herb being tested for treatment of Alzheimer's.

Immune Scavengers target plaques.

Meta-Study Yields 13 Prime Risk Genes But disease extends beyond gene predictors.

Brain Receptor May Play Role in Alzheimer's Defects in astrocyte formation could contribute to disease.

Enzyme Traced to Two Alzheimer's Pathways Loss of Pin1 implicated in development of both tangles and plaques.

Vaccine May Clear Alzheimer's Brain Plaques

Alzheimer's Plaques Reversed in Mice by Blocking Cholesterol Pathway

Alzheimer's Study
Maps Alternate Route to Disease.

Alzheimer's Culprit Fingered as Gang of Four Proteins responsible for Alzheimer's enzyme gamma-secretase identified.

>More Research Reports

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Last updated July 2007
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